The operational definition of mestizo by Pons-Estel et al. For the detection of the p. This study is part of the prospective study which we have conducted to analyze the salient features of the tromphophilic conditions in Mexican mestizos. Abnormal results for platelet aggregation with three concentrations of ADP 2. The method described by Mammen [ 6 ] was used: Individuals born in Latin America who had both Amerindian and white ancestors. Top 10 algorithms in data mining. 
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A decision tree was constructed using nine subsets, expressed as an easily interpretable rule sets, and cross-validated with the hold-out subset.
hwmatologia For the detection of the p. Acquired activated protein C resistance cause by lupus anticoagulants. Briefly, a fragment of the prothrombin gene comprising nucleotides 19,—20, was amplified by PCR. Top 10 algorithms in data mining. Am J Clin Pathol.
The detection of the transition c. This process was repeated permutating the attributes until each attribute had been selected once as target attribute.
J Arguelles
In this study we have analyzed some of the causes, both inherited and acquired, of the aPCR phenotype in a group of Mexican mestizos with a history of a thrombotic episode in the presence of a clinical marker of thrombophilia. National Center for Biotechnology InformationU. In a single institution for a month period, 96 Mexican mestizos with a history of thrombosis and hematoolgia markers of a primary thrombophilic state were prospectively studied to identify a thrombophilic condition.
Author information Article notes Copyright and License information Disclaimer. Data Analysis The highly efficient algorithm C4. Grupo Latinoamericano de Estudio de Lupus: Morgan Kaufmann Publishers; Functional abnormalities of this pathway, which manifest itself in vitro as a poor anticoagulant response of plasma to added APC aPC resistance, aPCR are prevalent in the general population and are associated with an increased risk of fundamentso thrombosis.
Effects of the oral, direct factor Xa inhibitor rivaroxaban on commonly used coagulation assays.
Please review our privacy policy. These data are consonant with those observed in other populations and further identify that consideration be given to identifying whether individuals are being treated with the novel DOACs when conducting laboratory studies oriented to identify the etiology of thrombosis.
Normal controls hemtologia studied for each case.
Hematologia Leucemia A by Angel Gamboa on Prezi
How we study and treat patients with suspected thrombophilia. Support Center Support Center.
Castoldi E, Rosing J. Individuals born in Latin America who had both Amerindian and white ancestors.
This study is part of the prospective study which we have conducted to analyze the salient features of the tromphophilic conditions in Mexican mestizos. The rest of the variables studied were not found to be associated with the presence of the aPCR phenotype. The accuracy of the classification was recorded. Abnormal results for platelet aggregation with three concentrations of ADP 2.
As a result of studying these conditions in fundamentso than Mexican mestizos, we have been able to define the thrombophilic conditions prevailing in the Mexican population, which express differences to those identified in other populations of the world [ 4 ].
The operational definition of mestizo by Pons-Estel et al. This article has been cited by other articles in PMC. Furthermore, autoimmune diseases predicted each other, i. Interestingly, when looking at the rule sets fundqmentos aPCR phenotype, in all cases only the status of FV Leiden mutation had been considered relevant for the classification. Materials and Methods Patients During a month period, all consecutive Mexican mestizo patients referred to our clinic by physicians from different parts of the country were prospectively accrued in the study, if they had one of the following clinical markers associated with a primary hypercoagulable state [ 1 — 4 ]: The method described by Mammen [ 6 ] was used: To look for the factor V gene Hong Kong mutation p.
Linkage between inherited resistance to activated protein C and factor V gene mutation in venous thrombosis.
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